Lifeboat Foundation

Giacomo Grasselli — a senior Italian government health official who is coordinating the network of intensive care units in Lombardy — explains the “critical” situation in Italy, brought about by the Covid-19 outbreak (Subscribe: https://bit.ly/C4_News_Subscribe)

——

Continue reading “Coronavirus ‘worse than a bomb’ on Italy, says doctor coordinating response” »

The Earth, solar system, the entire Milky Way and the few thousand galaxies closest to us move in a vast “bubble” that is 250 million light years in diameter, where the average density of matter is half as high as for the rest of the universe. This is the hypothesis advanced by a theoretical physicist from the University of Geneva (UNIGE) to solve a conundrum that has been splitting the scientific community for a decade: At what speed is the universe expanding? Until now, at least two independent calculation methods have arrived at two values that are different by about 10% with a deviation that is statistically irreconcilable. This new approach, which is set out in the journal Physics Letters B, erases this divergence without making use of any “new physics.”

The universe has been expanding since the Big Bang occurred 13.8 billion years ago—a proposition first made by the Belgian canon and physicist Georges Lemaître (1894−1966), and first demonstrated by Edwin Hubble (1889−1953). The American astronomer discovered in 1929 that every galaxy is pulling away from us, and that the most distant galaxies are moving the most quickly. This suggests that there was a time in the past when all the galaxies were located at the same spot, a time that can only correspond to the Big Bang. This research gave rise to the Hubble-Lemaître law, including the Hubble constant (H0), which denotes the universe’s rate of expansion. The best H0 estimates currently lie around 70 (km/s)/Mpc (meaning that the universe is expanding 70 kilometers a second more quickly every 3.26 million light years). The problem is that there are two conflicting methods of calculation.

Coronavirus Spread Spurs Debate Over Which Health Workers Need To Be Quarantined : Shots — Health News Should “potential exposure” by a health worker to someone with coronavirus be enough to send that worker home for two weeks of self-quarantine? Health systems have begun debating relative risks.

This month, an international team put all of those ingredients together, turning theory into reality.

The three labs, scattered across Padova, Italy, Zurich, Switzerland, and Southampton, England, collaborated to create a fully self-controlled, hybrid artificial-biological neural network that communicated using biological principles, but over the internet.

The three-neuron network, linked through artificial synapses that emulate the real thing, was able to reproduce a classic neuroscience experiment that’s considered the basis of learning and memory in the brain. In other words, artificial neuron and synapse “chips” have progressed to the point where they can actually use a biological neuron intermediary to form a circuit that, at least partially, behaves like the real thing.

We need to figure out the ethical implications before they arrive.

NASA’s Ames Research Center in California has issued a mandatory policy for employees to work from home after one worker tested positive for the coronavirus responsible for COVID-19.

The research center, which is located in Moffett Field in the Silicon Valley, has been placed on restricted access after the employee was confirmed to have the coronavirus on Sunday (March 8).

https://youtube.com/watch?v=QKi5T4uTGXk

Check your blood glucose without fingersticks using the FreeStyle Libre System, a continuous glucose monitoring system that includes a sensor and reader.

Scientists have found that an area of our brain, known as the Default Mode Network, is responsible for our ego and the subsequent psychological disorders that stem from it.

Circa 2018 we need crispr pills for this.

The steady march of aging might not have to be so steady, according to a growing body of research. Now a team from the University at Buffalo has isolated a single gene that controls senescence, the process that stops cells from dividing and contributes to aging symptoms. Ramping it up, they found how easily the effect can spread among neighboring cells. That makes the gene a crucial target for future work into anti-aging and cancer therapies.

Living cells have a natural limit to the amount of times they can divide, before they stop and become what are known as senescent cells. The problem is, over time these senescent cells build up in the body, eventually contributing to the physical symptoms we associate with aging, such as increased risk of diabetes, heart disease, arthritis and cataracts. On the other hand though, completely halting senescence can lead to cancer, as the cells continue to divide unchecked.

Continue reading “Scientists single out gene that triggers aging symptoms” »