Will humans soon live forever? Scientists believe it’s possible — and it could happen as early as 2050. In this video, we explore 10 shocking scientific breakthroughs that are pushing humanity closer to immortality. From nanobots that cure disease from within, to brain uploading, cloning organs, and AI-driven consciousness — this is the future of life itself.
🧬 Get ready to discover the jaw-dropping technologies that might just make death optional.
⚠️ Don’t blink. The future is coming faster than you think.
Macquarie University researchers have discovered a naturally occurring protein found in human cells plays a powerful role in repairing damaged DNA — the molecule that carries the genetic instructions for building and maintaining living things.
The discovery, published in the journalAgeing Cell, could hold the key to developing therapies for devastating age-related diseases such as motor neuron disease (MND), Alzheimer’s disease, and Parkinson’s disease.
Hope: Dr Sina Shadfar, pictured, and colleagues discovered a protein which they have shown for the first time acts like a ‘glue’, helping to repair broken DNA, which is widely accepted as one of the main contributors to ageing and the progression of age-related diseases.
The research, conducted by neurobiologist Dr Sina Shadfar and colleagues in the Motor Neuron Disease Research Centre, reveals a protein called protein disulphide isomerase (PDI) helps repair serious deoxyribonucleic acid (DNA) damage. This breakthrough opens new possibilities for therapies aimed at boosting the body’s ability to fix its own DNA — a process that becomes less efficient as we age.
Does Nature Obey Laws? | Sheldrake-Vernon Dialogue 95.
The conviction that the natural world is obedient, adhering to laws, is a widespread assumption of modern science. But where did this idea originate and what beliefs does it imply? In this episode of the Sheldrake-Vernon Dialogues, Rupert Sheldrake and Mark Vernon discuss the impact on science of the Elizabethan lawyer, Francis Bacon. His New Instrument of Thought, or Novum Organum, put laws at the centre of science and was intended as an upgrade on assumptions developed by Aristotle. But does the existence of mind-like laws of nature, somehow acting on otherwise mindless matter, even make sense? What difference is made by insights subsequent to Baconian philosophy, such as the discovery of evolution or the sense that the natural world is not machine-like but behaves like an organism? Could the laws of nature be more like habits? And what about the existence of miracles, the purposes of organisms, and the extraordinary fecundity of creativity?
— Dr Rupert Sheldrake, PhD, is a biologist and author best known for his hypothesis of morphic resonance. At Cambridge University, as a Fellow of Clare College, he was Director of Studies in biochemistry and cell biology. As the Rosenheim Research Fellow of the Royal Society, he carried out research on the development of plants and the ageing of cells, and together with Philip Rubery discovered the mechanism of polar auxin transport. In India, he was Principal Plant Physiologist at the International Crops Research Institute for the Semi-Arid Tropics, where he helped develop new cropping systems now widely used by farmers. He is the author of more than 100 papers in peer-reviewed journals and his research contributions have been widely recognized by the academic community, earning him a notable h-index for numerous citations. On ResearchGate his Research Interest Score puts him among the top 4% of scientists.
The future of human longevity just got a $500billion booste thanks to Trump’s bold new AI vision! In this video, we explore the recently announced Stargate AI Infrastructure Project—a staggering $500 billion initiative unveiled by President Donald Trump on January 21, 2025, alongside tech titans OpenAI, Oracle, and SoftBank. Designed to build colossal data centers and supercharge AI development, could this ambitious plan hold the key to curing aging? We dive into the cutting-edge science, the potential of AI to revolutionize healthcare, and how Stargate might reshape humanity’s fight against time. Buckle up for a wild ride into the future!
🔍 What You’ll Discover: The Stargate Project: Trump’s massive AI infrastructure gamble. How AI could unlock breakthroughs in aging research. The players: OpenAI, Oracle, SoftBank, and their bold vision. The big question: Can this tech triumph over mortality?
🔔 Like, subscribe, and hit the bell for more groundbreaking insights every week! Tell us in the comments: Do you think Stargate could be the key to curing aging? Let’s debate it!
Scientists are looking at ways to tackle Alzheimer’s and dementia from all kinds of angles, and a new study has identified the molecule hevin (or SPARCL-1) as a potential way of preventing cognitive decline.
Hevin is a protein naturally produced in the brain by cells called astrocytes. These support-worker cells look after the connections or synapses between neurons, and it’s thought that hevin plays a role in this essential work.
In this new study, researchers from the Federal University of Rio de Janeiro (UFRJ) and the University of São Paulo in Brazil boosted hevin production in the brains of both healthy mice and those with an Alzheimer’s-like disease.
Cells are constantly subjected to DNA damage from a range of internal and environmental sources. It is estimated that cells can experience as many as 100,000 DNA lesions per day. One of the most deleterious types of DNA lesions is the DNA double-strand break (DSB). Just one unrepaired DNA DSB may be enough to cause mutations or cell death leading to a wide range of pathologies including cancer, immune deficiency, premature aging and neurodegeneration.
To respond to the array of DNA lesions that occur, cells have developed a complex and coordinated series of steps involving DNA damage recognition, cell cycle arrest and signaling-induced activation of the DNA repair machinery—processes collectively referred to as the DNA damage response (DDR). In recent years, progress has been made in understanding how this process is initiated. However, the later stages of this process, including long range DNA end-resection, are not well understood.
In a new study published in Nature Cell Biology, researchers from Boston University Chobanian & Avedisian School of Medicine, Massachusetts General Hospital (MGH) and Harvard Medical School, identified several uncharacterized chromatin factors (proteins that regulate gene expression) that are recruited to sites of DNA damage, including the gene ZNF280A. Importantly, this gene is hemizygously deleted—meaning one of the two copies of alleles is missing—in a subset of patients with a human developmental syndrome called 22q11.2 distal deletion syndrome.
Hey! If anyone’s interested in attending the Viva Frontier Tower Longevity Summit in SF this weekend (Aubrey de Grey and Irina Conboy, plus a ton of others, are speaking) I have a couple 60% coupons I can share. Shoot me a DM!
On June 22–23, the Longevity Summit hosted during the 6-week Viva Frontier Tower Pop-up Village (Jun 20 — Aug 4) will serve two purposes:
