Videos will be released step by step over the next few weeks as we receive clearance from the individual speakers.
This week we kick it off with Jerry Shay, who is the Vice Chairman of the Department of Cell Biology at The University of Texas Southwestern Medical Center in Dallas, presenting ‘Telomeres and Telomerase in Aging and Cancer‘.
undoing-aging.org/…/jerry-shay-presenting-at-undoing-aging-…
More info on Forever Healthy: forever-healthy.org
ETH researchers have integrated two CRISPR-Cas9-based core processors into human cells. This represents a huge step towards creating powerful biocomputers.
Controlling gene expression through gene switches based on a model borrowed from the digital world has long been one of the primary objectives of synthetic biology. The digital technique uses what are known as logic gates to process input signals, creating circuits where, for example, output signal C is produced only when input signals A and B are simultaneously present.
To date, biotechnologists had attempted to build such digital circuits with the help of protein gene switches in cells. However, these had some serious disadvantages: they were not very flexible, could accept only simple programming, and were capable of processing just one input at a time, such as a specific metabolic molecule. More complex computational processes in cells are thus possible only under certain conditions, are unreliable, and frequently fail.
There have been a number of efforts to increase genome diversity. In 2010, the US National Institutes of Health (NIH) and the Wellcome Trust in London launched the Human Heredity and Health in Africa (H3Africa) initiative, which supports Africa-led genome research. And last year, the NIH started enrolment for the All of Us research programme, which plans to collect DNA and health data from hundreds of thousands of people of varying ethnicities in the United States.
Researchers from under-represented groups are making genomics more inclusive by working with communities that have been overlooked or abused.
A physics-based, “atavistic” model posits that cancer is a “safe mode” for stressed cells and suggests that oxygen and immunotherapy are the best ways to beat the disease.
Dangerous airborne viruses are rendered harmless on-the-fly when exposed to energetic, charged fragments of air molecules, University of Michigan researchers have shown.
They hope to one day harness this capability to replace a century-old device: the surgical mask.
The U-M engineers have measured the virus-killing speed and effectiveness of nonthermal plasmas—the ionized, or charged, particles that form around electrical discharges such as sparks. A nonthermal plasma reactor was able to inactivate or remove from the airstream 99.9% of a test virus, with the vast majority due to inactivation.
Researchers at Ben-Gurion University of the Negev (BGU) have discovered that gene mutations that once helped humans survive may increase the possibility for diseases, including cancer.
The findings were recently the cover story in the journal Genome Research.
The team of researchers from BGU’s National Institute for Biotechnology in the Negev (NIBN) set out to look for mutations in the genome of the mitochondria, a part of every cell responsible for energy production that is passed exclusively from mothers to their children. The mitochondria are essential to every cell’s survival and our ability to perform the functions of living.
News-Medical speaks to David Dambman from Biosero about the emerging importance of automation in scientific research and how a centralized scheduling software is an essential first step for any laboratory looking to automate their workflow.
Why has automation become so critical to advancing scientific research?
There are many reasons why automation is useful in scientific research. First and foremost, automation is about being able to walk away from your experiments and spend time analyzing your results, rather than carrying out mundane tasks such as transferring liquids from one plate to another.
